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Students asked inspired or challenging questions to all presenters and it was clear that considerable thought was put into synthesis of the questions. Questions by non-presenting group to the presenters on presentation attached Your presentation was very interesting and we were hoping you could elaborate a little more on your statement: the 1993 Study demonstrated the prevalence of diabetes in offspring, as well as obesity. We recognize that there are multiple forms of diabetes with different mechanisms and risk factors. The three most common types include: Type 1 (juvenile/autoimmune/insulin-dependent) diabetes: typically onsets in childhood, is highly heritable, and results from a complete absence of insulin in the body due to the destruction of beta-cells Type 2 (non-insulin dependent) diabetes: most common, typically onsets much later in adulthood as a result of insulin-resistance that has developed due to diets high in glucose/carbohydrates Gestational diabetes mellitus: intolerance to glucose as a result of a hormone produced by the placenta, often resolves after delivery; obesity is a risk factor but not always present Question: From what you have learned, how do you think the in utero mechanisms that predispose the fetus to either type 1 (childhood) or type 2 (adult onset) diabetes differ and might the risk factors be different for that fetus dependent on whether the mother has pre-existing/undiagnosed type 2 diabetes during pregnancy or gestational diabetes? For example, if we had an obese pregnant mother with pre-existing type 2 diabetes, how might her offsprings risk for future diabetes (either type 1 or type 2) differ from the offspring of another mother who was a healthy weight during pregnancy but developed gestational diabetes? -Ashley and Bria References: American Diabetes Association (2010). Diagnosis and classification of diabetes mellitus.
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